Azra Dad
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Research Participation Program/ ORISE Postdoctoral Fellow
Also affiliated: United States Food and Drug Administration (2017–2021); University of Illinois Urbana-Champaign (2013–2019); COMSATS University Islamabad (2013)
Postdoc Researcher
Research Areas
Biomedical Subjects
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Biography and Research Information
OverviewAI-generated summary
Azra Dad is a postdoctoral fellow at the National Center for Toxicological Research (NCTR) through the Research Participation Program/ORISE. Her research focuses on genotoxicity assessment, particularly investigating the effects of chemical mutagens in animal models. Dad has published work examining the dose-response relationship of ethyl methanesulfonate-induced genotoxicity across different life stages and tissues in rats. Her research also explores background mutation frequencies in cell lines such as TK6 and L5178Y, with implications for error-corrected sequencing.
Further contributions include studies on *Pig-a* gene mutations in the bone marrow granulocytes of rats treated with procarbazine. Her scholarly output includes 19 publications with 327 citations and an h-index of 10. Dad collaborates with researchers at NCTR, including Javier R. Revollo, Robert H. Heflich, and Mason G. Pearce, with whom she shares multiple publications.
Metrics
- h-index: 10
- Publications: 19
- Citations: 339
Selected Publications
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Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing (2025)
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Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity (2021)
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<i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats (2021)
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Molecular analysis of GPI-anchor biosynthesis pathway genes in rat strains used for the Pig-a gene mutation assay (2020)
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<scp>CD59</scp>‐deficient bone marrow erythroid cells from rats treated with procarbazine and propyl‐nitrosourea have mutations in the<i>Pig‐a</i>gene (2020)
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Fungicide suppression of flight performance in the honeybee ( <i>Apis mellifera</i> ) and its amelioration by quercetin (2019)
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Pig-a mutations in bone marrow erythroblasts of rats treated with 7,12-dimethyl-benz[a]anthracene (2019)
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Evaluation of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) mutagenicity using in vitro and in vivo Pig-a assays (2018)
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Analysis of mutation in the rat <i>Pig‐a</i> assay: I) studies with bone marrow erythroid cells (2018)
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Analysis of mutation in the rat <i>Pig‐a</i> assay: II. Studies with bone marrow granulocytes (2018)
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Genome-wide mutation detection by interclonal genetic variation (2018)
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Establishing a novel <i>Pig‐a</i> gene mutation assay in L5178Y<i>Tk</i><sup>+/−</sup> mouse lymphoma cells (2017)
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Spectrum of benzo[ a ]pyrene-induced mutations in the Pig-a gene of L5178Y Tk +/− cells identified with next generation sequencing (2017)
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Chloramination of wastewater effluent: Toxicity and formation of disinfection byproducts (2017)
Collaboration Network
Top Collaborators
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- <i>Pig‐a</i> gene mutations in bone marrow granulocytes of procarbazine‐treated <scp>F344</scp> rats
- Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity
- Effect of life stage and target tissue on dose–response assessment of ethyl methane sulfonate‐induced genotoxicity
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
- Background Mutation Frequencies in <scp>TK6</scp> and <scp>L5178Y</scp> Cells: Implications for Error‐Corrected Sequencing
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