Match tier
Confirmed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
P
Pieter Jan Koopmans
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Graduate Research Assistant
Also affiliated: Appalachian State University (2021–2023)
8 h-index
38 pubs
161 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Pieter Jan Koopmans' research investigates molecular responses to exercise and disease in skeletal muscle. His work explores the role of microRNAs (miRNAs) in adult skeletal muscle satellite cells and their control over the myogenic cell transcriptome and proteome, specifically examining miR-16 and miR-1.
Koopmans also studies the molecular landscape after exercise in humans, identifying MYC as a regulator of muscle growth. His research further examines the impact of mitochondrial antioxidants, such as SkQ1, on attenuating cancer-induced muscle wasting and improving muscle contractility in animal models. Additionally, his work addresses the intersection of epigenetics and senescence, investigating the effects of senolytic administration on DNA methylation clocks and the methylome in aged and regenerated skeletal muscle.
Metrics
- h-index: 8
- Publications: 38
- Citations: 161
Positions
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Graduate Research Assistant 2022–presentUniversity of Arkansas at Fayetteville Health, Human Performance, and Recreation ORCID
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Affiliate Research Faculty 2022–2032Appalachian State University Public Health and Exercise Science ORCID
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Graduate Research Assistant 2019–2022Appalachian State University Health and Exercise Science ORCID
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Specimen Processing Associate 2020–2021EXACT Sciences Corp Clinical Lab ORCID
Selected Publications
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Myonuclear Dynamics After Skeletal Muscle Surgical Injury (2026)
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Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations (2026)
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Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia (2026)
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Myonuclear Dynamics After Skeletal Muscle Surgical Injury (2026)
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Resident myonuclear adaptations to mechanical overload in single muscle fibers of HSA-GFP mice (2026)
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TRAIL: A potential new biomarker for muscle health in anorexia nervosa (2026)
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Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans (2026)
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Myonuclear Dynamics After Skeletal Muscle Surgical Resection Injury (2026)Journal of the Arkansas Academy of Science OpenAlex
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The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice (2026)
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Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle (2025)
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The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus (2025)
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Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle (2025)
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Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females (2025)
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Corrigendum to “microRNA-1 regulates metabolic flexibility by programming skeletal muscle pyruvate metabolism” [Mol Metabol 98 (2025) 1–23/102182] (2025)
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microRNA-1 regulates metabolic flexibility by programming adult skeletal muscle pyruvate metabolism (2025)
Collaboration Network
Top Collaborators
Kevin A. Murach
University of Arkansas at Fayetteville
23 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The roles of miRNAs in adult skeletal muscle satellite cells
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Going nuclear: Molecular adaptations to exercise mediated by myonuclei
- At the Nexus Between Epigenetics and Senescence: The Effects of Senolytic ( <scp>BI01</scp> ) Administration on <scp>DNA</scp> Methylation Clock Age and the Methylome in Aged and Regenerated Skeletal Muscle
Showing 5 of 23 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
9 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- At the Nexus Between Epigenetics and Senescence: The Effects of Senolytic ( <scp>BI01</scp> ) Administration on <scp>DNA</scp> Methylation Clock Age and the Methylome in Aged and Regenerated Skeletal Muscle
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
Showing 5 of 9 shared publications
Nicholas P. Greene
University of Arkansas at Fayetteville
9 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- At the Nexus Between Epigenetics and Senescence: The Effects of Senolytic ( <scp>BI01</scp> ) Administration on <scp>DNA</scp> Methylation Clock Age and the Methylome in Aged and Regenerated Skeletal Muscle
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
Showing 5 of 9 shared publications
Ana Regina Cabrera
University of Arkansas at Fayetteville
7 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
Showing 5 of 7 shared publications
Ronald G. Jones
University of Arkansas at Fayetteville
6 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
- Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans
Showing 5 of 6 shared publications
Ahmed Ismaeel
Auburn University (US)
6 shared publications
- The roles of miRNAs in adult skeletal muscle satellite cells
- microRNA-1 regulates metabolic flexibility by programming adult skeletal muscle pyruvate metabolism
- The Age‐Dependent Resident Myonuclear Multi‐Omic Response to an Acute Skeletal Muscle Hypertrophic Stimulus in Mice
- microRNA-1 Regulates Metabolic Flexibility in Skeletal Muscle via Pyruvate Metabolism
- The Age-Dependent Resident Myonuclear Multi-Omic Response to a Skeletal Muscle Hypertrophic Stimulus
Showing 5 of 6 shared publications
Sabin Khadgi
University of Arkansas at Fayetteville
6 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Showing 5 of 6 shared publications
Calvin Peterson
University of Arkansas at Fayetteville
6 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
Showing 5 of 6 shared publications
Toby L. Chambers
University of Arkansas at Fayetteville
6 shared publications
In database
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- At the Nexus Between Epigenetics and Senescence: The Effects of Senolytic ( <scp>BI01</scp> ) Administration on <scp>DNA</scp> Methylation Clock Age and the Methylome in Aged and Regenerated Skeletal Muscle
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans
Showing 5 of 6 shared publications
Nathan Serrano
University of Arkansas at Fayetteville
6 shared publications
In database
- Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle
- Displaced myonuclei are attributable to both resident myonuclear migration and stem cell fusion during mechanical loading in adult skeletal muscle
- Myonuclear Dynamics After Skeletal Muscle Surgical Resection Injury
- Myonuclear Dynamics After Skeletal Muscle Surgical Injury
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
Showing 5 of 6 shared publications
Ferdinand von Walden
Hong Kong Science and Technology Parks Corporation (HK)
5 shared publications
- The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
- The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates <i>MYC</i> as a Hypertrophic Regulator That is Sufficient for Growth
- Ribosome dynamics during skeletal muscle repair and regeneration in mice and humans
- Insight on the loading-mediated regulation of Runx1 in skeletal muscle
Stavroula Tsitkanou
Beth Israel Deaconess Medical Center
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
Ruqaiza Muhyudin
University of Arkansas at Fayetteville
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Exercise training prior to and during cancer in mice preserves muscle mass, reduces tumour weight and suppresses molecular mediators of cachexia
Seongkyun Lim
University of Arkansas at Fayetteville
4 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- MicroRNA control of the myogenic cell transcriptome and proteome: the role of miR-16
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
Katarzyna Goljanek‐Whysall
Ollscoil na Gaillimhe – University of Galway (IE)
4 shared publications
- The roles of miRNAs in adult skeletal muscle satellite cells
- microRNA-1 regulates metabolic flexibility by programming adult skeletal muscle pyruvate metabolism
- microRNA-1 Regulates Metabolic Flexibility in Skeletal Muscle via Pyruvate Metabolism
- Corrigendum to “microRNA-1 regulates metabolic flexibility by programming skeletal muscle pyruvate metabolism” [Mol Metabol 98 (2025) 1–23/102182]
Similar Researchers
Based on overlapping research topics
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